Entry - #101900 - ACROKERATOSIS VERRUCIFORMIS; AKV - OMIM
# 101900

ACROKERATOSIS VERRUCIFORMIS; AKV


Alternative titles; symbols

HOPF DISEASE


Phenotype-Gene Relationships

Location Phenotype Phenotype
MIM number
Inheritance Phenotype
mapping key
Gene/Locus Gene/Locus
MIM number
12q24.11 Acrokeratosis verruciformis 101900 AD 3 ATP2A2 108740
Clinical Synopsis
 

INHERITANCE
- Autosomal dominant
SKIN, NAILS, & HAIR
Skin
- Multiple, flat, skin-colored warty papules (2-4mm in diameter on dorsum hands, feet, knees, and elbows)
- Punctate keratoses (palms and soles)
Skin Histology
- Hyperkeratosis
- Acanthosis
- Papillomatosis
- Prominent granular cell layer
- No acantholysis or dyskeratosis
Nails
- Longitudinal red and white lines
- Nail fragility
- Distal V-shaped notching
- Longitudinal ridging
MISCELLANEOUS
- Hyperkeratosis often present at birth but may appear later
MOLECULAR BASIS
- Caused by mutation in the ATPase, Ca++ transporting, slow-twitch, cardiac muscle-2 gene (ATP2A2, 108740)

TEXT

A number sign (#) is used with this entry because of evidence that acrokeratosis verruciformis (AKV) is caused by heterozygous mutation in the ATP2A2 gene (108740) on chromosome 12q24.

Darier-White disease (DAR; 124200) is also caused by mutation in the ATP2A2 gene.


Description

Acrokeratosis verruciformis of Hopf (AKV) is a localized disorder of keratinization affecting the distal extremities. Onset occurs early in life (Dhitavat et al., 2003).


Clinical Features

In the family with acrokeratosis verruciformis of Hopf reported by Niedelman (1947) and Niedelman and McKusick (1962), the dorsum of the hands was affected first and most conspicuously. Older individuals tended to have hyperkeratosis of the elbows and knees. The nails were pearly white in early years and become horny, brown, ridged, and grooved in later life. In older affected persons the palms and soles became involved.

Although histology of acrokeratosis verruciformis lesions shows no evidence of dyskeratosis, Herndon and Wilson (1966) emphasized the phenotypic overlap between this entity and Darier-White disease (124200) and even proposed that they may not be separate entities. In the family they studied, 7 persons had typical acrokeratosis verruciformis, 1 or possibly 2 had Darier disease, and 3 had minor disturbances of keratinization (white nails from subungual hyperkeratosis, or punctate keratoses of palms or soles). Also see benign familial pemphigus (169600).


Inheritance

The pedigree with acrokeratosis verruciformis studied by Niedelman (1947) and Niedelman and McKusick (1962) contained instances of male-to-male transmission as well as unaffected daughters of affected males, consistent with autosomal dominant inheritance.


Molecular Genetics

Dhitavat et al. (2003) studied a family with acrokeratosis verruciformis in 6 generations and identified a heterozygous missense mutation in the ATP2A2 gene (P602L; 108740.0011). This mutation predicted a nonconservative amino acid substitution in the ATP binding domain of the molecule. The mutation segregated with the disease phenotype in the family and was not found in 50 controls. Moreover, functional analysis of the P602L mutant showed that it had lost its ability to transport Ca(2+). This result demonstrated loss of function of the sarco(endo)plasmic reticulum Ca(2+) ATPase2 mutant in acrokeratosis verruciformis, thus providing evidence that acrokeratosis verruciformis and Darier disease are allelic disorders.

In an 11-year-old Afghan boy with AKV, Berk et al. (2010) identified a heterozygous missense mutation in the ATP2A2 gene (A698V; 108740.0013).


REFERENCES

  1. Berk, D. R., Taube, J. M., Bruckner, A. L., Lane, A. T. A sporadic patient with acrokeratosis verruciformis of Hopf and a novel ATP2A2 mutation. (Letter) Brit. J. Derm. 163: 653-654, 2010. [PubMed: 20518781, related citations] [Full Text]

  2. Dhitavat, J., Macfarlane, S., Dode, L., Leslie, N., Sakuntabhai, A., MacSween, R., Saihan, E., Hovnanian, A. Acrokeratosis verruciformis of Hopf is caused by mutation in ATP2A2: evidence that it is allelic to Darier's disease. J. Invest. Derm. 120: 229-232, 2003. [PubMed: 12542527, related citations] [Full Text]

  3. Herndon, J. H., Jr., Wilson, J. D. Acrokeratosis verruciformis (Hopf) and Darier's disease: genetic evidence for a unitary origin. Arch. Derm. 93: 305-310, 1966.

  4. Niedelman, M. L., McKusick, V. A. Acrokeratosis verruciformis (Hopf): a follow-up study. Arch. Derm. 86: 779-782, 1962. [PubMed: 13938495, related citations] [Full Text]

  5. Niedelman, M. L. Acrokeratosis verruciformis (Hopf): report of fourteen cases in one family in four generations, with a review of the literature. Arch. Derm. Syph. 56: 48-63, 1947. [PubMed: 20251558, related citations] [Full Text]


Contributors:
Gary A. Bellus - updated : 5/1/2003
Creation Date:
Victor A. McKusick : 6/4/1986
carol : 07/10/2024
carol : 11/02/2022
carol : 09/01/2021
terry : 03/27/2012
terry : 3/20/2012
carol : 3/20/2012
carol : 3/19/2012
alopez : 5/1/2003
alopez : 2/10/2003
mark : 4/19/1995
pfoster : 9/2/1994
mimadm : 3/11/1994
supermim : 3/16/1992
carol : 8/23/1990
supermim : 3/20/1990

# 101900

ACROKERATOSIS VERRUCIFORMIS; AKV


Alternative titles; symbols

HOPF DISEASE


SNOMEDCT: 400085009;   ORPHA: 79151;   DO: 0050606;  


Phenotype-Gene Relationships

Location Phenotype Phenotype
MIM number
Inheritance Phenotype
mapping key
Gene/Locus Gene/Locus
MIM number
12q24.11 Acrokeratosis verruciformis 101900 Autosomal dominant 3 ATP2A2 108740

TEXT

A number sign (#) is used with this entry because of evidence that acrokeratosis verruciformis (AKV) is caused by heterozygous mutation in the ATP2A2 gene (108740) on chromosome 12q24.

Darier-White disease (DAR; 124200) is also caused by mutation in the ATP2A2 gene.


Description

Acrokeratosis verruciformis of Hopf (AKV) is a localized disorder of keratinization affecting the distal extremities. Onset occurs early in life (Dhitavat et al., 2003).


Clinical Features

In the family with acrokeratosis verruciformis of Hopf reported by Niedelman (1947) and Niedelman and McKusick (1962), the dorsum of the hands was affected first and most conspicuously. Older individuals tended to have hyperkeratosis of the elbows and knees. The nails were pearly white in early years and become horny, brown, ridged, and grooved in later life. In older affected persons the palms and soles became involved.

Although histology of acrokeratosis verruciformis lesions shows no evidence of dyskeratosis, Herndon and Wilson (1966) emphasized the phenotypic overlap between this entity and Darier-White disease (124200) and even proposed that they may not be separate entities. In the family they studied, 7 persons had typical acrokeratosis verruciformis, 1 or possibly 2 had Darier disease, and 3 had minor disturbances of keratinization (white nails from subungual hyperkeratosis, or punctate keratoses of palms or soles). Also see benign familial pemphigus (169600).


Inheritance

The pedigree with acrokeratosis verruciformis studied by Niedelman (1947) and Niedelman and McKusick (1962) contained instances of male-to-male transmission as well as unaffected daughters of affected males, consistent with autosomal dominant inheritance.


Molecular Genetics

Dhitavat et al. (2003) studied a family with acrokeratosis verruciformis in 6 generations and identified a heterozygous missense mutation in the ATP2A2 gene (P602L; 108740.0011). This mutation predicted a nonconservative amino acid substitution in the ATP binding domain of the molecule. The mutation segregated with the disease phenotype in the family and was not found in 50 controls. Moreover, functional analysis of the P602L mutant showed that it had lost its ability to transport Ca(2+). This result demonstrated loss of function of the sarco(endo)plasmic reticulum Ca(2+) ATPase2 mutant in acrokeratosis verruciformis, thus providing evidence that acrokeratosis verruciformis and Darier disease are allelic disorders.

In an 11-year-old Afghan boy with AKV, Berk et al. (2010) identified a heterozygous missense mutation in the ATP2A2 gene (A698V; 108740.0013).


REFERENCES

  1. Berk, D. R., Taube, J. M., Bruckner, A. L., Lane, A. T. A sporadic patient with acrokeratosis verruciformis of Hopf and a novel ATP2A2 mutation. (Letter) Brit. J. Derm. 163: 653-654, 2010. [PubMed: 20518781] [Full Text: https://doi.org/10.1111/j.1365-2133.2010.09876.x]

  2. Dhitavat, J., Macfarlane, S., Dode, L., Leslie, N., Sakuntabhai, A., MacSween, R., Saihan, E., Hovnanian, A. Acrokeratosis verruciformis of Hopf is caused by mutation in ATP2A2: evidence that it is allelic to Darier's disease. J. Invest. Derm. 120: 229-232, 2003. [PubMed: 12542527] [Full Text: https://doi.org/10.1046/j.1523-1747.2003.t01-1-12045.x]

  3. Herndon, J. H., Jr., Wilson, J. D. Acrokeratosis verruciformis (Hopf) and Darier's disease: genetic evidence for a unitary origin. Arch. Derm. 93: 305-310, 1966.

  4. Niedelman, M. L., McKusick, V. A. Acrokeratosis verruciformis (Hopf): a follow-up study. Arch. Derm. 86: 779-782, 1962. [PubMed: 13938495] [Full Text: https://doi.org/10.1001/archderm.1962.01590120077013]

  5. Niedelman, M. L. Acrokeratosis verruciformis (Hopf): report of fourteen cases in one family in four generations, with a review of the literature. Arch. Derm. Syph. 56: 48-63, 1947. [PubMed: 20251558] [Full Text: https://doi.org/10.1001/archderm.1947.01520070051005]


Contributors:
Gary A. Bellus - updated : 5/1/2003

Creation Date:
Victor A. McKusick : 6/4/1986

Edit History:
carol : 07/10/2024
carol : 11/02/2022
carol : 09/01/2021
terry : 03/27/2012
terry : 3/20/2012
carol : 3/20/2012
carol : 3/19/2012
alopez : 5/1/2003
alopez : 2/10/2003
mark : 4/19/1995
pfoster : 9/2/1994
mimadm : 3/11/1994
supermim : 3/16/1992
carol : 8/23/1990
supermim : 3/20/1990