Entry - #610093 - MICROPHTHALMIA, ISOLATED 2; MCOP2 - OMIM
# 610093

MICROPHTHALMIA, ISOLATED 2; MCOP2


Alternative titles; symbols

ANOPHTHALMIA, CLINICAL, ISOLATED


Phenotype-Gene Relationships

Location Phenotype Phenotype
MIM number
Inheritance Phenotype
mapping key
Gene/Locus Gene/Locus
MIM number
14q24.3 Microphthalmia, isolated 2 610093 AR 3 VSX2 142993
Clinical Synopsis
 
Phenotypic Series
 

INHERITANCE
- Autosomal recessive
HEAD & NECK
Eyes
- Microphthalmia
- Short palpebral fissures
- Corneal opacity
MOLECULAR BASIS
- Caused by mutation in the visual system homeobox 2 gene (VSX2, 142993.0003)

TEXT

A number sign (#) is used with this entry because isolated microphthalmia-2 (MCOP2) is caused by homozygous mutation in the CHX10 gene (142993) on chromosome 14q24.

For a phenotypic description and a discussion of genetic heterogeneity of isolated microphthalmia, see MCOP1 (251600).


Nomenclature

The term 'anophthalmia' has been misused in the medical literature. True or primary anophthalmia is rarely compatible with life; in such cases, the primary optic vesicle has stopped developing and the abnormal development involves major defects in the brain as well (Francois, 1961). The diagnosis can only be made histologically (Reddy et al., 2003; Morini et al., 2005; Smartt et al., 2005), but this is rarely done. In most published cases, the term 'anophthalmia' is used as a synonym for the more appropriate terms 'extreme microphthalmia' or 'clinical anophthalmia.'


Clinical Features

Kohn et al. (1988) described a large consanguineous Arab kindred in Israel in which 19 persons over 3 generations had bilateral profound microphthalmia without associated anomalies and with normal intelligence. One female infant had tracheoesophageal fistula repaired in the neonatal period; when examined at 1 month of age, she had mild right hydronephrosis by intravenous pyelography and ultrasound. Kohn et al. (1988) stated that these abnormalities were 'undoubtedly unrelated to the microphthalmia' (see microphthalmia and esophageal atresia, MCOPS3, 206900).


Molecular Genetics

Bar-Yosef et al. (2004) studied 4 families (2 of Arab origin, 1 of Bedouin origin, and 1 of Persian Jewish origin) with autosomal recessive isolated microphthalmia/clinical anophthalmia and no associated eye anomalies. In the 2 Arab families, 1 of which had previously been reported by Kohn et al. (1988), they identified homozygosity for a missense mutation (142993.0003) and a deletion (142993.0004) in the CHX10 gene, respectively. No mutations were identified in the other 2 families.

In 6 affected individuals from 2 consanguineous families from Qatar with isolated microphthalmia and cloudy corneas, Faiyaz-Ul-Haque et al. (2007) identified homozygosity for an R200P mutation (142993.0002) in the CHX10 gene. Craniofacial features, height, weight, and intelligence were normal in these patients. Unaffected parents were heterozygous for the mutation.


REFERENCES

  1. Bar-Yosef, U., Abuelaish, I., Harel, T., Hendler, N., Ofir, R., Birk, O. S. CHX10 mutations cause non-syndromic microphthalmia/anophthalmia in Arab and Jewish kindreds. Hum. Genet. 115: 302-309, 2004. [PubMed: 15257456, related citations] [Full Text]

  2. Faiyaz-Ul-Haque, M., Zaidi, S. H. E., Al-Mureikhi, M. S., Peltekova, I., Tsui, L.-C., Teebi, A. S. Mutations in the CHX10 gene in nonsyndromic microphthalmia/anophthalmia patients from Qatar. Clin. Genet. 72: 164-166, 2007. [PubMed: 17661825, related citations] [Full Text]

  3. Francois, J. Heredity in Ophthalmology. St. Louis: C. V. Mosby (pub.) 1961. P. 173.

  4. Kohn, G., El Shawwa, R., El Rayyes, E. Isolated 'clinical anophthalmia' in an extensively affected Arab kindred. Clin. Genet. 33: 321-324, 1988. [PubMed: 3378363, related citations]

  5. Morini, F., Pacilli, M., Spitz, L. Bilateral anophthalmia and esophageal atresia: report of a new patient and review of the literature. (Letter) Am. J. Med. Genet. 132A: 60-62, 2005. [PubMed: 15389708, related citations] [Full Text]

  6. Reddy, M. A., Francis, P. J., Berry, V., Bradshaw, K., Patel, R. J., Maher, E. R., Kumar, R., Bhattacharya, S. S., Moore, A. T. A clinical and molecular genetic study of a rare dominantly inherited syndrome (MRCS) comprising of (sic) microcornea, rod-cone dystrophy, cataract, and posterior staphyloma. Brit. J. Ophthal. 87: 197-202, 2003. [PubMed: 12543751, images, related citations] [Full Text]

  7. Smartt, J. M., Jr., Kherani, F., Saddiqi, F., Katowitz, J. A., Bartlett, S. P. Microphthalmia and synostotic frontal plagiocephaly: a rare clinical entity with implications for craniofacial reconstruction. Plast. Reconstr. Surg. 116: 1e-9e, 2005. Note: Electronic Article. [PubMed: 15988238, related citations] [Full Text]


Contributors:
Marla J. F. O'Neill - updated : 10/30/2007
Creation Date:
Marla J. F. O'Neill : 5/3/2006
mcolton : 08/07/2015
carol : 2/26/2014
mcolton : 2/26/2014
wwang : 10/30/2007
carol : 5/17/2007
carol : 5/7/2007
carol : 6/1/2006
carol : 5/3/2006

# 610093

MICROPHTHALMIA, ISOLATED 2; MCOP2


Alternative titles; symbols

ANOPHTHALMIA, CLINICAL, ISOLATED


ORPHA: 2542;   DO: 0060839;  


Phenotype-Gene Relationships

Location Phenotype Phenotype
MIM number
Inheritance Phenotype
mapping key
Gene/Locus Gene/Locus
MIM number
14q24.3 Microphthalmia, isolated 2 610093 Autosomal recessive 3 VSX2 142993

TEXT

A number sign (#) is used with this entry because isolated microphthalmia-2 (MCOP2) is caused by homozygous mutation in the CHX10 gene (142993) on chromosome 14q24.

For a phenotypic description and a discussion of genetic heterogeneity of isolated microphthalmia, see MCOP1 (251600).


Nomenclature

The term 'anophthalmia' has been misused in the medical literature. True or primary anophthalmia is rarely compatible with life; in such cases, the primary optic vesicle has stopped developing and the abnormal development involves major defects in the brain as well (Francois, 1961). The diagnosis can only be made histologically (Reddy et al., 2003; Morini et al., 2005; Smartt et al., 2005), but this is rarely done. In most published cases, the term 'anophthalmia' is used as a synonym for the more appropriate terms 'extreme microphthalmia' or 'clinical anophthalmia.'


Clinical Features

Kohn et al. (1988) described a large consanguineous Arab kindred in Israel in which 19 persons over 3 generations had bilateral profound microphthalmia without associated anomalies and with normal intelligence. One female infant had tracheoesophageal fistula repaired in the neonatal period; when examined at 1 month of age, she had mild right hydronephrosis by intravenous pyelography and ultrasound. Kohn et al. (1988) stated that these abnormalities were 'undoubtedly unrelated to the microphthalmia' (see microphthalmia and esophageal atresia, MCOPS3, 206900).


Molecular Genetics

Bar-Yosef et al. (2004) studied 4 families (2 of Arab origin, 1 of Bedouin origin, and 1 of Persian Jewish origin) with autosomal recessive isolated microphthalmia/clinical anophthalmia and no associated eye anomalies. In the 2 Arab families, 1 of which had previously been reported by Kohn et al. (1988), they identified homozygosity for a missense mutation (142993.0003) and a deletion (142993.0004) in the CHX10 gene, respectively. No mutations were identified in the other 2 families.

In 6 affected individuals from 2 consanguineous families from Qatar with isolated microphthalmia and cloudy corneas, Faiyaz-Ul-Haque et al. (2007) identified homozygosity for an R200P mutation (142993.0002) in the CHX10 gene. Craniofacial features, height, weight, and intelligence were normal in these patients. Unaffected parents were heterozygous for the mutation.


REFERENCES

  1. Bar-Yosef, U., Abuelaish, I., Harel, T., Hendler, N., Ofir, R., Birk, O. S. CHX10 mutations cause non-syndromic microphthalmia/anophthalmia in Arab and Jewish kindreds. Hum. Genet. 115: 302-309, 2004. [PubMed: 15257456] [Full Text: https://doi.org/10.1007/s00439-004-1154-2]

  2. Faiyaz-Ul-Haque, M., Zaidi, S. H. E., Al-Mureikhi, M. S., Peltekova, I., Tsui, L.-C., Teebi, A. S. Mutations in the CHX10 gene in nonsyndromic microphthalmia/anophthalmia patients from Qatar. Clin. Genet. 72: 164-166, 2007. [PubMed: 17661825] [Full Text: https://doi.org/10.1111/j.1399-0004.2007.00846.x]

  3. Francois, J. Heredity in Ophthalmology. St. Louis: C. V. Mosby (pub.) 1961. P. 173.

  4. Kohn, G., El Shawwa, R., El Rayyes, E. Isolated 'clinical anophthalmia' in an extensively affected Arab kindred. Clin. Genet. 33: 321-324, 1988. [PubMed: 3378363]

  5. Morini, F., Pacilli, M., Spitz, L. Bilateral anophthalmia and esophageal atresia: report of a new patient and review of the literature. (Letter) Am. J. Med. Genet. 132A: 60-62, 2005. [PubMed: 15389708] [Full Text: https://doi.org/10.1002/ajmg.a.30283]

  6. Reddy, M. A., Francis, P. J., Berry, V., Bradshaw, K., Patel, R. J., Maher, E. R., Kumar, R., Bhattacharya, S. S., Moore, A. T. A clinical and molecular genetic study of a rare dominantly inherited syndrome (MRCS) comprising of (sic) microcornea, rod-cone dystrophy, cataract, and posterior staphyloma. Brit. J. Ophthal. 87: 197-202, 2003. [PubMed: 12543751] [Full Text: https://doi.org/10.1136/bjo.87.2.197]

  7. Smartt, J. M., Jr., Kherani, F., Saddiqi, F., Katowitz, J. A., Bartlett, S. P. Microphthalmia and synostotic frontal plagiocephaly: a rare clinical entity with implications for craniofacial reconstruction. Plast. Reconstr. Surg. 116: 1e-9e, 2005. Note: Electronic Article. [PubMed: 15988238] [Full Text: https://doi.org/10.1097/01.prs.0000169706.29344.e4]


Contributors:
Marla J. F. O'Neill - updated : 10/30/2007

Creation Date:
Marla J. F. O'Neill : 5/3/2006

Edit History:
mcolton : 08/07/2015
carol : 2/26/2014
mcolton : 2/26/2014
wwang : 10/30/2007
carol : 5/17/2007
carol : 5/7/2007
carol : 6/1/2006
carol : 5/3/2006